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学者姓名：邵敬伟
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| GB/T 7714 | Tong, Ling-Wu , Le, Jing-Qing , Song, Xun-Huan et al. Synergistic anti-tumor effect of dual drug co-assembled nanoparticles based on ursolic acid and sorafenib (vol 234, 113724, 2024) [J]. | COLLOIDS AND SURFACES B-BIOINTERFACES , 2025 , 253 . |
| MLA | Tong, Ling-Wu et al. "Synergistic anti-tumor effect of dual drug co-assembled nanoparticles based on ursolic acid and sorafenib (vol 234, 113724, 2024)" . | COLLOIDS AND SURFACES B-BIOINTERFACES 253 (2025) . |
| APA | Tong, Ling-Wu , Le, Jing-Qing , Song, Xun-Huan , Li, Cheng-Lei , Yu, Shi-Jing , Lin, Ying-Qi et al. Synergistic anti-tumor effect of dual drug co-assembled nanoparticles based on ursolic acid and sorafenib (vol 234, 113724, 2024) . | COLLOIDS AND SURFACES B-BIOINTERFACES , 2025 , 253 . |
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Reprogramming the immunosuppressive tumor microenvironment (TME) to boost CD8+ T cell infiltration is crucial for anti-tumor immunotherapy. In this study, a bionic nanoplatform (VGRM) based on a two-dimensional vanadium carbide (MXene) carrying glucose oxidase (GOx) and CRISPR/Cas9 was constructed, which enabled a cascade reaction that amplified the generation of reactive oxygen species (ROS) while depleting glutathione (GSH) by combining MXenzyme and natural enzymes. The CRISPR/Cas9 system could reduce the expression of MTH1, depress tumor cells' self-defense against oxidative stress and thus significantly enhance the therapeutic effect of ROS. Additionally, the excellent NIR-II photothermal performance endowed VGRM with photoacoustic imaging (PA) and photothermal therapy (PTT) capabilities. The enhanced oxidative stress and photothermal killing ability could activate the cGAS/STING innate immune pathway, induce immunogenic cell death (ICD), and at the same time reverse the immunosuppressive TME, which promoted CD8+ T cells infiltration, and thereby inhibiting tumor proliferation. Meanwhile, antigen-activated memory T cells (adaptive immunity) could suppress tumor recurrence and metastasis effectively. This study provides a novel strategy for the combined application of MXene and gene editing therapy in modulating the tumor immune microenvironment.
Keyword ：
Adaptive immunity Adaptive immunity cGAS/STING cGAS/STING CRISPR/Cas9 CRISPR/Cas9 MXene MXene Theranostics Theranostics Tumor immune microenvironment Tumor immune microenvironment
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| GB/T 7714 | Feng, Ke-Ke , Li, Cheng-Lei , Tu, Yi-Fan et al. Multi-level ROS regulation to activate innate and adaptive immune therapies [J]. | CHEMICAL ENGINEERING JOURNAL , 2025 , 515 . |
| MLA | Feng, Ke-Ke et al. "Multi-level ROS regulation to activate innate and adaptive immune therapies" . | CHEMICAL ENGINEERING JOURNAL 515 (2025) . |
| APA | Feng, Ke-Ke , Li, Cheng-Lei , Tu, Yi-Fan , Tian, Shi-Cheng , Xiong, Rui , Sa, Bai-Sheng et al. Multi-level ROS regulation to activate innate and adaptive immune therapies . | CHEMICAL ENGINEERING JOURNAL , 2025 , 515 . |
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Immune checkpoint blockade (ICB) holds broad application prospects in tumor treatment. However, the poor immunogenicity and the adaptive resistance cells severely limit the clinical efficacy. Immunogenic cell death (ICD) can sensitize the cells which were initially lower responsive to ICB. Based on this, we reported a metal coordination co-assembly nanocomposite (SIF NPs), which can enhance cancer immunotherapy by strengthening ICD induction through inducing ferroptosis-photo-chemo. The SIF NPs possess excellent biocompatibility and stability, and exhibit good PTT and PDT effects both in vitro and in vivo. The SIF NPs disrupt redox homeostasis and induce ferroptosis by consuming glutathione (GSH), downregulating the expression of solute carrier family 7 member 11 (SLC7A11) and glutathione peroxidase 4 (GPX4), and enhancing the generation of lipid hydroperoxides (LPO). Importantly, SIF NPs trigger an ICD cascade via inducing the cell death, thus enhancing the immunogenicity of tumors. In a tumor-bearing mice model, the synergistic therapy of SIF NPs and anti-PD-L1 antibody demonstrated a powerful inhibition effectiveness on bilateral tumors and instigated a long-time immune memory effect. This study indicates that the combined treatment of ferroptosis-photo-chemo and immunotherapy possess promising application prospects.
Keyword ：
Ferroptosis-photo-chemo Ferroptosis-photo-chemo Immunogenic cell death Immunogenic cell death Immunotherapy Immunotherapy Synergistic therapy Synergistic therapy
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| GB/T 7714 | Li, Cheng-Lei , Zhang, Wen-Zhong , Zhao, Rui-Rui et al. Synergistic ferroptosis-photo-chemo to disrupt redox homeostasis for augmenting cancer chemoimmunotherapy [J]. | COLLOIDS AND SURFACES B-BIOINTERFACES , 2025 , 255 . |
| MLA | Li, Cheng-Lei et al. "Synergistic ferroptosis-photo-chemo to disrupt redox homeostasis for augmenting cancer chemoimmunotherapy" . | COLLOIDS AND SURFACES B-BIOINTERFACES 255 (2025) . |
| APA | Li, Cheng-Lei , Zhang, Wen-Zhong , Zhao, Rui-Rui , Le, Jing-Qing , Zhang, Bing-Chen , Xie, Huan-Zhang et al. Synergistic ferroptosis-photo-chemo to disrupt redox homeostasis for augmenting cancer chemoimmunotherapy . | COLLOIDS AND SURFACES B-BIOINTERFACES , 2025 , 255 . |
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Photodynamic therapy (PDT) has arisen as a promising method due to its spatiotemporal precision and minimal invasiveness. It encounters significant obstacles in solid tumors due to hypoxia-induced therapeutic resistance and the self-protective mechanisms of cancer cells facilitated by MutT homolog 1 (MTH1), an enzyme involved in oxidative damage repair. Herein, we fabricate a tumor-microenvironment responsive CRISPR nanoplatform based on hollow mesoporous manganese dioxide (H-MnO2) for PDT. This platform utilizes H-MnO2 to produce oxygen (O2) through the decomposition of hydrogen peroxide (H2O2) in TME, thereby mitigating hypoxia and enhancing reactive oxygen species (ROS) generation. The high concentration of glutathione (GSH) and hyaluronidase (HAase) in TME induces the release of CRISPR/Cas9 ribonucleoproteins (RNP) to target the MTH1 gene, thereby impairs oxidative damage repair pathways and amplifys ROS-mediated cytotoxicity. The released Mn2+ ions function as immunomodulatory agents, activate innate immune responses via stimulating STING signal pathway. In vitro, IHMRH NPs markedly increased intracellular O2 levels, ROS production, lipid peroxidation and DNA damage, leading to tumor cell death, immune activation, and effective gene editing. In vivo, the nanoplatform suppressed tumor growth, diminished MTH1 gene expression, stimulated dendritic cell (DC) maturation through immunogenic cell death (ICD). This multimodal nanosystem may amplifies oxidative stress,
Keyword ：
CRISPR/Cas9 CRISPR/Cas9 Immune activation Immune activation Photodynamic therapy Photodynamic therapy Synergy therapy Synergy therapy
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| GB/T 7714 | Tian, Shi-Cheng , Song, Xun-Huan , Feng, Ke-Ke et al. Self-oxygenating nanoplatform integrating CRISPR/Cas9 gene editing and immune activation for highly efficient photodynamic therapy [J]. | JOURNAL OF COLLOID AND INTERFACE SCIENCE , 2025 , 693 . |
| MLA | Tian, Shi-Cheng et al. "Self-oxygenating nanoplatform integrating CRISPR/Cas9 gene editing and immune activation for highly efficient photodynamic therapy" . | JOURNAL OF COLLOID AND INTERFACE SCIENCE 693 (2025) . |
| APA | Tian, Shi-Cheng , Song, Xun-Huan , Feng, Ke-Ke , Li, Cheng-Lei , Tu, Yi-Fan , Hu, Yong-Shan et al. Self-oxygenating nanoplatform integrating CRISPR/Cas9 gene editing and immune activation for highly efficient photodynamic therapy . | JOURNAL OF COLLOID AND INTERFACE SCIENCE , 2025 , 693 . |
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The existing therapies for cancer are not remote satisfactory due to drug-resistance in tumors that are malignant. There is a pressing necessity to take a step forward to develop innovative therapies that can complement current ones. Multiple investigations have demonstrated that ferroptosis therapy, a non-apoptotic modality of programmed cell death, has tremendous potential in face of multiple crucial events, such as drug resistance and toxicity in aggressive malignancies. Recently, ferroptosis at the crosswalk of chemotherapy, materials science, immunotherapy, tumor microenvironment, and bionanotechnology has been presented to elucidate its therapeutic feasibility. Given the burgeoning progression of ferroptosis-based nanomedicine, the newest advancements in this field at the confluence of ferroptosis-inducers, nanotherapeutics, along with tumor microenvironment are given an overview. Here, the signaling pathways of ferroptosis-related were first talked about briefly. The emphasis discussion was placed on the pharmacological mechanisms and the nanodrugs design of ferroptosis inducing agents based on multiple distinct metabolism pathways. Additionally, a comprehensive overview of the action mechanisms by which the tumor microenvironment influences ferroptosis was elaborately descripted. Finally, some limitations of current researches and future research directions were also deliberately discussed to provide details about therapeutic avenues for ferroptosis-related diseases along with the design of anti-drugs.
Keyword ：
Ferroptosis Ferroptosis Nanodrug design Nanodrug design Pathway Pathway Pharmacological mechanism Pharmacological mechanism Tumor microenvironment Tumor microenvironment
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| GB/T 7714 | Yu, Shijing , Tong, Lingwu , Shen, Jiangwen et al. Recent research progress based on ferroptosis-related signaling pathways and the tumor microenvironment on it effects [J]. | EUROPEAN JOURNAL OF MEDICINAL CHEMISTRY , 2024 , 269 . |
| MLA | Yu, Shijing et al. "Recent research progress based on ferroptosis-related signaling pathways and the tumor microenvironment on it effects" . | EUROPEAN JOURNAL OF MEDICINAL CHEMISTRY 269 (2024) . |
| APA | Yu, Shijing , Tong, Lingwu , Shen, Jiangwen , Li, Chenglei , Hu, Yongshan , Feng, Keke et al. Recent research progress based on ferroptosis-related signaling pathways and the tumor microenvironment on it effects . | EUROPEAN JOURNAL OF MEDICINAL CHEMISTRY , 2024 , 269 . |
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Ferroptosis, an emerging cell death mode discovered in recent decades, is characterized by lipid peroxidation during cell death, usually manifested as iron accumulation. However, the forthright delivery of iron species will cause adverse detrimental effects such as anaphylactic reactions in routine tissues. So far, studies on cellular ferroptosis by employing bio-derived nanomaterials have rarely been acquired. Herein, we reported carbon dots (CDs)-based biocompatible enzymes with negligible toxicity from a representative hepatoprotective triterpenoid natural product ursolic acid (UA) with acknowledged antitumor activity. We observed that CDs presented distinct GSH oxidase-like characteristics and then contributed ferroptosis of carcinoma cells by interfering with the GPX4-catalyzed lipid repair system. After the CDs were assembled with UA, the obtained denoted as UCDs integrated multi-model therapy into one, resulting in enhanced therapeutic efficacy, reduced adverse effects, and optimized clinical outcomes. In vivo, the UCDs hysterically inhibited tumor growth in hepatoma H22-bearing mice with inconsequential toxicity. Exceptionally, UCDs enlisted a large number of tumor-infiltrating immune cells, including T cells, NK cells, and macrophages in H22 tumor-bearing mice, consequently transforming "cold" into "hot" tumors to activate systemic anti-tumor immune responses. Meanwhile, UCDs could also awfully suppress the H22-LUC tumor growth in orthotopic xenograft mouse models. Collectively, our research commends that the drug delivery systems based on natural-products-derived CDs can function as biocompatible enzymes for antitumor treatment and may facilitate the advancement of nanotechnology-based immunotherapeutics. It is extremely anticipated that such ferroptosis-like designing in nano-catalytic medicine would be favorable for future development in cancer-therapeutic regimens.
Keyword ：
Ferroptosis Ferroptosis Hepatocellular carcinoma Hepatocellular carcinoma Nanozyme Nanozyme Tumor immune microenvironment Tumor immune microenvironment
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| GB/T 7714 | Lai, Chun-Mei , Xu, Jia , Zhang, Bing -Chen et al. A natural product-derived nanozyme regulator induced chemo-ferroptosis dual therapy in remodeling of the tumor immune microenvironment of hepatocellular carcinoma [J]. | CHEMICAL ENGINEERING JOURNAL , 2024 , 482 . |
| MLA | Lai, Chun-Mei et al. "A natural product-derived nanozyme regulator induced chemo-ferroptosis dual therapy in remodeling of the tumor immune microenvironment of hepatocellular carcinoma" . | CHEMICAL ENGINEERING JOURNAL 482 (2024) . |
| APA | Lai, Chun-Mei , Xu, Jia , Zhang, Bing -Chen , He, Shao-Hua , Shao, Jing-Wei . A natural product-derived nanozyme regulator induced chemo-ferroptosis dual therapy in remodeling of the tumor immune microenvironment of hepatocellular carcinoma . | CHEMICAL ENGINEERING JOURNAL , 2024 , 482 . |
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Liver cancer, which is well-known to us as one of human most prevalent malignancies across the globe, poses a significant risk to live condition and life safety of individuals in every region of the planet. It has been shown that immune checkpoint treatment may enhance survival benefits and make a significant contribution to patient prognosis, which makes it a promising and popular therapeutic option for treating liver cancer at the current time. However, there are only a very few numbers of patients who can benefit from the treatment and there also exist adverse events such as toxic effects and so on, which is still required further research and discussion. Fortunately, the clustered regularly interspaced short palindromic repeat/CRISPR-associated nuclease 9 (CRISPR/Cas9) provides a potential strategy for immunotherapy and immune checkpoint therapy of liver cancer. In this review, we focus on elucidating the fundamentals of the recently developed CRISPR/Cas9 technology as well as the present-day landscape of immune checkpoint treatment which pertains to liver cancer. What's more, we aim to explore the molecular mechanism of immune checkpoint treatment in liver cancer based on CRISPR/Cas9 technology. At last, its encouraging and powerful potential in the future application of the clinic is discussed, along with the issues that already exist and the difficulties that must be overcome. To sum up, our ultimate goal is to create a fresh knowledge that we can utilize this new CRISPR/Cas9 technology for the current popular immune checkpoint therapy to overcome the treatment issues of liver cancer.
Keyword ：
CRISPR/Cas9 CRISPR/Cas9 immune checkpoint immune checkpoint immunotherapy immunotherapy liver cancer liver cancer
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| GB/T 7714 | Tong, Ling-Wu , Hu, Yong-Shan , Yu, Shi-Jing et al. Current application and future perspective of CRISPR/cas9 gene editing system mediated immune checkpoint for liver cancer treatment [J]. | NANOTECHNOLOGY , 2024 , 35 (40) . |
| MLA | Tong, Ling-Wu et al. "Current application and future perspective of CRISPR/cas9 gene editing system mediated immune checkpoint for liver cancer treatment" . | NANOTECHNOLOGY 35 . 40 (2024) . |
| APA | Tong, Ling-Wu , Hu, Yong-Shan , Yu, Shi-Jing , Li, Cheng-Lei , Shao, Jing-Wei . Current application and future perspective of CRISPR/cas9 gene editing system mediated immune checkpoint for liver cancer treatment . | NANOTECHNOLOGY , 2024 , 35 (40) . |
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Both ursolic acid (UA) and sorafenib (Sora) have been generally utilized in cancer treatment, and the combination of the two has also shown a good anti-tumor effect. However, single-agent therapy for Hepatocellular carcinoma (HCC) has the disadvantages of multi-drug resistance, poor water solubility and low bioavailability, and the application of traditional nanocarrier materials is limited due to their low drug loading and low carrierrelated toxicity. Therefore, we prepared US NPs with different proportions of UA and Sora by solvent exchange method for achieving synergistic HCC therapy. US NPs had suitable particle size, good dispersibility and storage stability, which synergistically inhibited the proliferation of HepG2 cells, SMMC7721 cells and H22 cells. In addition, we also proved that US NPs were able to suppress the migration of HepG2 cells and SMMC7721 cells and reduce the adhesion ability and colony formation ability of these cells. According to the results, US NPs could degrade the membrane potential of mitochondrial, participate in cell apoptosis, and synergistically induce autophagy. Collectively, the carrier-free US NPs provide new strategies for HCC treatment and new ideas for the development of novel nano-drug delivery systems containing UA and Sora.
Keyword ：
Co-assembly Co-assembly Sorafenib Sorafenib Synergistic therapy Synergistic therapy Ursolic acid Ursolic acid
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| GB/T 7714 | Tong, Ling-Wu , Le, Jing-Qing , Song, Xun-Huan et al. Synergistic anti-tumor effect of dual drug co-assembled nanoparticles based on ursolic acid and sorafenib [J]. | COLLOIDS AND SURFACES B-BIOINTERFACES , 2024 , 234 . |
| MLA | Tong, Ling-Wu et al. "Synergistic anti-tumor effect of dual drug co-assembled nanoparticles based on ursolic acid and sorafenib" . | COLLOIDS AND SURFACES B-BIOINTERFACES 234 (2024) . |
| APA | Tong, Ling-Wu , Le, Jing-Qing , Song, Xun-Huan , Li, Cheng-Lei , Yu, Shi-Jing , Lin, Ying-Qi et al. Synergistic anti-tumor effect of dual drug co-assembled nanoparticles based on ursolic acid and sorafenib . | COLLOIDS AND SURFACES B-BIOINTERFACES , 2024 , 234 . |
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The heterogeneity of hepatocellular carcinoma (HCC) and the complexity of the tumor microenvironment (TME) pose challenges to efficient drug delivery and the antitumor efficacy of combined or synergistic therapies. Herein, a metal-coordinated carrier-free nanodrug (named as USFe3+ LA NPs) was developed for ferroptosis-mediated multimodal synergistic anti-HCC. Natural product ursolic acid (UA) was incorporated to enhance the sensitivity of tumor cells to sorafenib (SRF). Surface decoration of cell penetration peptide and epithelial cell adhesion molecule aptamer facilitated the uptake of USFe3+ LA NPs by HepG2 cells. Meanwhile, Fe3+ ions could react with intracellular hydrogen peroxide, generating toxic hydroxyl radical (& sdot;OH) for chemodynamical therapy (CDT) and amplified ferroptosis by cystine/glutamate antiporter system (System Xc ), which promoted the consumption of glutathione (GSH) and inhibited the expression of glutathione peroxidase 4 (GPX4). Notably, these all-in-one nanodrugs could inhibit tumor metastasis and induced immunogenic cell death (ICD). Last but not least, the nanodrugs demonstrated favorable biocompatibility, augmenting the immune response against the programmed death-ligand 1 (PD-L1) by increasing cytotoxic T cell infiltration. In vivo studies revealed significant suppression of tumor growth and distant metastasis. Overall, our work introduced a novel strategy for applications of metalcoordinated co-assembled carrier-free nano-delivery system in HCC combination therapy, especially in the realms of cancer metastasis prevention and immunotherapy.
Keyword ：
Chemodynamical therapy Chemodynamical therapy Ferroptosis Ferroptosis Immunotherapy Immunotherapy Metal -coordinated nanoplatform Metal -coordinated nanoplatform Synergistic chemotherapy Synergistic chemotherapy
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| GB/T 7714 | Zhao, Rui-Rui , Wu, Ju-Hong , Li, Jin -Yu et al. Multifunctional metal-coordinated Co-assembled carrier-free nanoplatform based on dual-drugs for ferroptosis-mediated cocktail therapy of hepatocellular carcinoma growth and metastasis [J]. | JOURNAL OF COLLOID AND INTERFACE SCIENCE , 2024 , 660 : 257-276 . |
| MLA | Zhao, Rui-Rui et al. "Multifunctional metal-coordinated Co-assembled carrier-free nanoplatform based on dual-drugs for ferroptosis-mediated cocktail therapy of hepatocellular carcinoma growth and metastasis" . | JOURNAL OF COLLOID AND INTERFACE SCIENCE 660 (2024) : 257-276 . |
| APA | Zhao, Rui-Rui , Wu, Ju-Hong , Li, Jin -Yu , Lu, Yu-sheng , Shao, Jing -Wei . Multifunctional metal-coordinated Co-assembled carrier-free nanoplatform based on dual-drugs for ferroptosis-mediated cocktail therapy of hepatocellular carcinoma growth and metastasis . | JOURNAL OF COLLOID AND INTERFACE SCIENCE , 2024 , 660 , 257-276 . |
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Combination immunotherapy has shown promising potential for enhancing the objective response rate compared to immune checkpoint blockade (ICB) monotherapy. However, combination therapy with multi-drugs is limited by the different properties of the agents and inconsistent synergistic targeted delivery. Herein, based on a universal triterpene template and the anticancer active agent ursolic acid (UA), a cytomembrane-coated biomimetic delivery nanoplatform (UR@M) prepared by the selfassembly of a PD-L1 targeted CRISPR/Cas9 system and UA was designed for hepatocellular carcinoma (HCC) treatment. UR@M showed enhanced tumor accumulation in vivo with homologous tumor targeting, and CRISPR in the nanosystem exhibited potent gene-editing efficiency of 76.53% in vitro and 62.42% in vivo with no off-target effects. UA activated the natural immune system through the TLR2-MyD88-TRAF6 pathway, which synergistically enhanced the proliferation of natural killer cells and dendritic cells and realized excellent immune cytotoxic T cell infiltration by combining with the ICB of PD-L1. The strategy of work along both lines based on innate immune and adaptive immunity displayed a significant effect in tumor regression. Overall, the UA-templated strategy "killed three birds with one stone" by establishing a self-assembly nanosystem, inducing tumor cell death, and promoting synergistic immunostimulation for HCC treatment.
Keyword ：
Biomimetic nanoplatform Biomimetic nanoplatform CRISPR/Cas9 CRISPR/Cas9 Gene therapy Gene therapy Hepatocellular carcinoma Hepatocellular carcinoma Immune checkpoint blockade Immune checkpoint blockade Immunotherapy Immunotherapy Self-assembly Self-assembly Ursolic acid Ursolic acid
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| GB/T 7714 | Zhang, Bing-Chen , Lai, Chun-Mei , Luo, Bang-Yue et al. Triterpenoids-templated self-assembly nanosystem for biomimetic delivery of CRISPR/ Cas9 based on the synergy of TLR-2 and ICB to enhance HCC immunotherapy [J]. | ACTA PHARMACEUTICA SINICA B , 2024 , 14 (7) : 3205-3217 . |
| MLA | Zhang, Bing-Chen et al. "Triterpenoids-templated self-assembly nanosystem for biomimetic delivery of CRISPR/ Cas9 based on the synergy of TLR-2 and ICB to enhance HCC immunotherapy" . | ACTA PHARMACEUTICA SINICA B 14 . 7 (2024) : 3205-3217 . |
| APA | Zhang, Bing-Chen , Lai, Chun-Mei , Luo, Bang-Yue , Shao, Jing-Wei . Triterpenoids-templated self-assembly nanosystem for biomimetic delivery of CRISPR/ Cas9 based on the synergy of TLR-2 and ICB to enhance HCC immunotherapy . | ACTA PHARMACEUTICA SINICA B , 2024 , 14 (7) , 3205-3217 . |
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